!!! *** New !!!New:
the new Local authority Care Actlink to carer page..Fact sheet 8 law for Carers *** New !!!

A Local Authority official descibes the new duties towards carers e.g....clause10,20;42-47 duty of safe guarding , 67-68 advocacy, 72 appealing ....

Section 117 aftercare

SchizophreniaWatch :- July 2014:

.... in your most need to be by your side ... Everyman

A website, principally for carers about and carers for schizophrenia; family, community, and professional service caring, who should - must ! - give voice for the best interests of those cared for they can't, and don't, voice for themselves.

The site is edited by a retired psychiatrist who has looked after someone with the schizophrenia condition, at home, affected by the negative form of schizophrenia, for the last twenty years. [The blue highlit links take you to another page. ] You return via the back arrow at the top of your screen or from a link at the bottom of a page


contact me if you want to help me out with my views, email about some issue of caring service that is promising, or unsettled and unsettling : at email me! or, or text to 07581384722.

There is a limited general psychiatry section links

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Section 1 Carer understanding of Schizophrenia.

What is the change in the brain that allows schizophrenia to come about.. To explain the cause(s) of schizophrenia

How to guide behaviour towards some recovery that is based on an understanding of those changes at the start of the illness.

.Section 2. Dealing with the NHS Services

Care in the first illness The caring journey

Carer Dealings with the Mental Health Trust 'Teams'

NEW and Important




Carers and those who live day to day with the illness have no doubts that it is in an illness.
There is a break change in observed behaviour from before, placing the person in alien behaviour, to their own disadvantage, which they are unable to reverse by themselves to regain the previous state.
The illness continues on a course with the same pattern, as an independent system, isolating the person from others, who puzzle out the changed behaviour by using their own experience to understand it.
It is a mental illness, arising out of a disabling change in the structure and function of the brain, the organ of the mind .

Unlike other medical illnesses the brain is removed from immediate access without the risk of damage. There can be no biopsy , no test for the change at the time of the onset of the illness. This has been the biggest challenge to accepting schizophrenia as an illness.
The diagnosis is made from the observation by family of odd behaviour . That behaviour is not always seen in the structured professional interview by someone who is a stranger to the sufferer.

There is the ability to measure a change in the brain - but at one step removed - by functional MRI scanning. Verifying and agreeing conclusions from these studies doesn't give a clear direction to diagnosis.

More persuasive result can come from brain material examined in post mortem tissue examination.

It depends upon family carers who are nearest relatives, giving permission to have brains removed - usually it would arise at Coroner Inquest after accidental death or suicide.( at this moment it is essential to confirm the findings of Reif et al 2006 .... neurogenesis in the hippocampus is not changed in depression - but is in schizophrenia ] for particular examination of the new cell formation areas. Plenty of material is available for the post-mortem study of Alzhemer's.
Generally, much fewer 'donations' come from those with schizophrenia. They are not encouraged by professionals, nor by family.

Families are put off by a lack of instruction about the the procedures involved, by the effect it might have upon funeral arrangements, practical and emotional within the family obligations.

The absence of Brain tissue to study is hampering research that would better explain the illness, would make it better undisquiet derstood, would lessen the stigma that comes with a fear from ignorance about what to do.


It is a common observation by family and clinicians that at the age at which schizophrenia starts - that is where their lives remain.
At the age when schizophrenia arrives that is when the accumulation of further experience stops.


Over the past two decades, several studies have shown that short-lived new nerve cells [ neurogenesis ] are continuously generated in the hippocampus area of the brain
In animal studies, disrupting nerve cell generation resulted in the loss of memory function, whilst increasing the production of new nerve cells led to improved memory. In 2006 a study found reduced new cell activity in the hippocampus .... Reif et al 2006

In schizophrenia the hippocampus area of the brain either is not carrying enough stem cells; or, it is not turning stem cells into enough new cells to provide enough new cells to support working memory


Will this new study help?


Earlier work had showed that young blood could stimulate the growth of brain stem cells and new neurons, as well as work that indicated that giving old blood to young mice can have the opposite effect, impairing their cognitive abilities. The young mice were 3 months old; the elderly mice were 18 months old.

The UCSF and Stanford scientists also directly injected old mice with young-mouse blood plasma, the yellowish liquid base of blood in which proteins and other solids are suspended. Over the course of three weeks, the old mice received eight blood plasma injections from young mice. Afterward, the treated mice remembered how to find a hidden resting platform in a water maze better than the controls did. They also exhibited better recollection of a chamber they had been conditioned to associate with a mild foot shock.

While the ingredient in the young blood responsible for these effects is still unknown, a clue was provided when the scientists heated the plasma before injection, and no such benefits were seen. Since proteins are deactivated by heat, the results are consistent with the relevant circulating factor being a protein.

The protein used in the study, called GDF11, was already known to reduce age-related heart enlargement, which is characteristic of heart failure. But Wagers said the new work shows that GDF11 has a similar age-reversal effect on other tissue, in particular the skeletal muscle and brain.

Links to study References

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